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Publikation · PMID 34542150

GWAS meta-analysis followed by Mendelian randomization revealed potential control mechanisms for circulating α-Klotho levels

Gergei I, Zheng J, Andlauer TFM, Brandenburg V, Mirza-Schreiber N, Müller-Myhsok B, Krämer BK, Richard D et al.

A GWAS meta-analysis of circulating alpha-Klotho levels in 4,376 individuals from LURIC and ALSPAC identified six genome-wide significant signals at five loci (ABO, KL, FGFR1, B4GALNT3, CHST9) explaining more than 9% of trait variance. Mendelian randomization indicated causal effects of Crohn’s disease liability (IVW beta = 0.059) and LDL cholesterol (IVW beta = -0.198) on alpha-Klotho, implicating post-translational modification enzymes and pathways beyond phosphate homeostasis in its regulation.

Human Molecular Genetics, 3. März 2022 | PMID 34542150

Zeitschrift
Human Molecular Genetics
DOI
10.1093/hmg/ddab263
PMID
34542150
Quelle
https://pubmed.ncbi.nlm.nih.gov/34542150/
Zitationen
19 Relative Zitationsrate 1.89 (1,0 = Durchschnitt des Fachgebiets) · NIH iCite, Stand 16.08.2026